Thanks, Carolyne, for your appreciation and for "keeping your eyes peeled".

In a way we are sweating the small stuff, as you say, but not in terms of a drug that may be too weak. Rather in terms of not addressing the main cause of CVD: too little exercise and too much of the wrong foods. In a word, lifestyle. For Lp(a) to inflict damage, the health and integrity of the arterial endothelium needs to be compromised. If an unfavorable lifestyle has caused this "clingwrap lining" to become porous, so to speak, LDL and Lp(a) may easily migrate through these cracks and accumulate as atherosclerotic lesions. So, yes, I think it may make sense to get tested for it, but whether that test will make a difference depends on the individual's motivation to do something to improve their risk profile.